Inflammatory cytokine
[대역어] None
[용어속성] Protein
[용어속성] Protein
3D Ex vivo tissue platforms to investigate the early phases of influenza a virus- and SARS-CoV-2-induced respiratory diseases
Article
Published on
Journal: Emerging Microbes & Infections
[Category] COVID19(2023년), SARS, 신약개발, 진단, 치료제,
Journal: Emerging Microbes & Infections
[Category] COVID19(2023년), SARS, 신약개발, 진단, 치료제,
[키워드] animal model
animal models
Antiviral
Cell
Cell culture
comparable
consequence
Drug repurposing
drug target
early phase
Ex vivo
ex vivo infection
Ex vivo infection model
Health
Host
human lung
IAV
in vitro
Infection
Inflammatory cytokine
Influenza
influenza A virus
Influenza virus
Innate immunity
innate immunity.
interferon
interferon response
Intervention
introduced
lung tissue
morbidity and mortality
murine
outbreak
pathophysiological
pharmacological
platform
Respiratory disease
respiratory viruses
responses
SARS-CoV-2
therapy
tissue
Treatment
viral infection
viral life cycle
viral replication
virus
virus
while
[DOI] 10.1080/22221751.2022.2117101 PMC 바로가기
[DOI] 10.1080/22221751.2022.2117101 PMC 바로가기
Single-cell transcriptomic atlas reveals distinct immunological responses between COVID-19 vaccine and natural SARS-CoV-2 infection
Article
[키워드] Activation
acute respiratory syndrome
alteration
analysis
antibody production
Apoptosis
B cell
caused
collected
CoronaVac
coronavirus
COVID-19
COVID-19 infected patient
COVID-19 patients
COVID-19 vaccine
cytotoxicity
downregulation
Emergency use authorization
expression
expression of gene
feature
greater
HLA class II
IL21R
immune
Immune cell
immune cell types
immune response
immunization
immunized
immunological
Immunological response
immunological responses
inactivated
Inactivated vaccine
individual
individuals
Infection
Inflammatory cytokine
innate immune cell
interferon response
magnitude
mechanism
Migration
mononuclear cells
naïve B cell
naïve B cells
pandemic
PBMC
Peripheral blood
phenotype
plasma
profile
Proinflammatory cytokine
proportion
provide
reveal
SARS-CoV-2
SARS-COV-2 infection
scRNA-seq
Sequencing
severe disease
single-cell
Single-cell RNA sequencing
single-cell sequencing.
Sinovac
systemic immune response
T cell
thrombosis
transcriptomic
upregulation
vaccine-induced immune response
[DOI] 10.1002/jmv.28012 PMC 바로가기
[DOI] 10.1002/jmv.28012 PMC 바로가기
B.1.351 SARS-CoV-2 Variant Exhibits Higher Virulence but Less Viral Shedding than That of the Ancestral Strain in Young Nonhuman Primates
Article
[키워드] addition
Alpha
animal model
association
B.1.351
Beta
caused
changes in
Characteristics
Chinese
circulating
Clinical signs
contribute
determine
distribution
drugs
early stage
Efficacy and safety
elevated
exhibited
IFN-γ
IL-17
Infection
Inflammatory
Inflammatory cytokine
interferon gamma
interleukin 17
investigated
less
Lower respiratory tract
lung tissue
mechanisms
mutated
Necrosis
NHP
NHPs
nonhuman primate
pathogenesis of SARS-CoV-2
pathogenic
pathogenicity
pathological changes
primate
provide
pulmonary inflammation
reduced
replicated
Research
rhesus macaque
SARS-CoV-2
SARS-CoV-2 strain
SARS-CoV-2 vaccine
SARS-CoV-2 variant
SARS-CoV-2 variants
severe lung injury
shedding
spike-specific T-cell response
strain
Strains
tissue
TNF-α
Transmission
upper respiratory tract
Vaccine
variant
variants of concern
viral distribution
viral shedding
virulence
virulence.
virulent
VoC
VOCs
were infected
[DOI] 10.1128/spectrum.02263-22 PMC 바로가기
[DOI] 10.1128/spectrum.02263-22 PMC 바로가기
Immunological and Pathological Peculiarity of Severe Acute Respiratory Syndrome Coronavirus 2 Beta Variant
Article
[키워드] Activation
acute respiratory syndrome
analysis
Beta
Beta variant
blockade
cascade
cause
CCL2
Characteristics
Comparative analysis
coronavirus
coronavirus 2
COVID-19
COVID-19 pandemic
CXCR2
cytokine
cytokine release
death
Disease progression
early stage
evidenced
IL-1β
immunological
induce
Infection
Inflammatory cytokine
investigated
K18-hACE2
late stage
Lung pathology
mice
molecular mechanism
Mortality
neutrophil recruitment
outbreak
pathological
pathological condition
pathology
phenotype
presymptomatic
provide
reduced
respiratory
SARS-CoV-2
SARS-COV-2 infection
SARS-CoV-2 pathogenesis
SARS-CoV-2 variant
secreted cytokine
Strains
the SARS-CoV-2
Transcriptome
transcriptomes
Transmissibility
Treatment
variant
Variant.
viral infection
weight loss
[DOI] 10.1128/spectrum.02371-22 PMC 바로가기
[DOI] 10.1128/spectrum.02371-22 PMC 바로가기
Can iron, zinc, copper and selenium status be a prognostic determinant in COVID-19 patients?
Review article
Published on
Journal: Environmental toxicology and pharmacology
[Category] COVID19(2023년), SARS, 바이오마커, 변종,
Journal: Environmental toxicology and pharmacology
[Category] COVID19(2023년), SARS, 바이오마커, 변종,
[키워드] Can
Concentration
Control
copper
correlated
COVID-19
COVID-19 patients
death rate
deficiency
dispersal
exhibit
facilitate
host immune responses
increase in
independent risk factor
Infection
Inflammatory cytokine
iron
modify
outcome
oxidative stress
Patient
Prognosis
prognostic
regulate
replication of SARS-CoV-2
risk of infections
SARS-CoV-2
Selenium
serum
serum Cu levels
severe COVID-19
significantly higher
Spike protein
suppress
the SARS-CoV-2
viral genome
viral replication
while
worse prognosis
Zinc
[DOI] 10.1016/j.etap.2022.103937 [Article Type] Review article
[DOI] 10.1016/j.etap.2022.103937 [Article Type] Review article
Identification of serum metabolites enhancing inflammatory responses in COVID-19
Article
[키워드] activate
Activation
acute respiratory syndrome
Administered
agmatine
alleviate
Antiviral
caused
changed
characterized
circulating metabolite
coronavirus
Coronavirus disease 2019
COVID-19
COVID-19 infection
COVID-19 patient
COVID-19 patients
cytokine production
drug
Effect
effector
elevated
elicit
embryonic
exhibiting
fibroblast
identification
IFNs
IL-6
IL-6 level
Inflammation
Inflammatory cytokine
Inflammatory response
interferon regulatory factor
interferons
IRFs
macrophages
metabolite
metabolites
metabolites.
metabolome
mice
molecular mechanism
NF-κB
NF-κB pathway
Proinflammatory cytokine
putrescine
responsible
resulting
SARS-CoV-2
serum
severity
significantly
stages
strong immune response
systemic inflammatory responses
Tandem mass spectrometry
tested
the disease
TNF
type I interferon
underlie
wildtype
[DOI] 10.1007/s11427-021-2099-7 PMC 바로가기
[DOI] 10.1007/s11427-021-2099-7 PMC 바로가기
Persistent but dysfunctional mucosal SARS-CoV-2-specific IgA and low lung IL-1β associate with COVID-19 fatal outcome: A cross-sectional analysis
Article
[키워드] acute respiratory syndrome
addition
Antibody Response
Antigen
BAL
Bronchoalveolar lavage
clearance
coronavirus
coronavirus disease
COVID-19
cross-sectional analysis
domain
fatal COVID-19
function
IgA
IgG
IL-1β
immune complex
Infection
Inflammatory
Inflammatory cytokine
lung
mucosal
mucosal immunity
neutralization
Neutralizing activity
Neutralizing titer
nucleocapsid protein
outcome
Patient
persistent
Protective
RBD
RBD variants
reduced
replicate
SARS-CoV-2
SARS-CoV-2-specific antibody
severe COVID-19 patients
Severe infection
severe infection.
Survivors
tested
viral clearance
virus
virus
virus elimination
with COVID-19
Wuhan
[DOI] 10.3389/fimmu.2022.842468 PMC 바로가기
[DOI] 10.3389/fimmu.2022.842468 PMC 바로가기
Prophylactic treatment of Glycyrrhiza glabra mitigates COVID-19 pathology through inhibition of pro-inflammatory cytokines in the hamster model and NETosis
Article
[키워드] accompanied
acute respiratory distress
addition
Comorbidity
coronavirus disease
COVID-19
COVID-19 pathology
cytokine production
decrease
dysregulated immune response
effective
Efficacy
Evidence
functions
glycyrrhiza
Glycyrrhiza glabra
Hamster model
hamsters
hypercoagulable
IL-17A
IL-4
immunocompromised individuals
immunomodulator
immunomodulatory properties
Inflammatory cytokine
ingredient
Lung pathology
lung viral load
mice
mice and human neutrophil functions.
mitigate
Mortality
mRNA expression
murine
NET
NETosis
neutrophil
polymorphonuclear neutrophil
pro-inflammatory cytokine
Protective
provide
Pulmonary pathology
reduced
reducing
robust
ROS
SARS-CoV-2
SARS-COV-2 infection
skewed
suppress
syndrome
Th17
Th2
Th2/Th17 differentiation
Treatment
vaccination
Vaccine
variant
[DOI] 10.3389/fimmu.2022.945583 PMC 바로가기
[DOI] 10.3389/fimmu.2022.945583 PMC 바로가기
Targeting Doublecortin-Like Kinase 1 (DCLK1)-Regulated SARS-CoV-2 Pathogenesis in COVID-19
Article
[키워드] Activation
amplified
amplify
analyzed
blocked
Blood
Cell
chemokine
chronic liver disease
Chronic Liver Diseases
circulating
comorbid disease
Comorbidity
contribute
controls
correlated
COVID-19
COVID-19 patients
Critical
cytokine
cytokine secretion
DCLK1
DDCLK1
decrease
disease severity
dysregulation
expression
Factor
Factors
Gain
growth factor
hospitalized patient
Host
Hyperinflammatory response
immune
Immune cell
immune response
immunosuppressive
in viral
in vitro
infected cells
Inflammation
Inflammatory cytokine
inflammatory pathway
inhibition
inhibitor
Innate immunity
involved
kinase
Live virus
liver
livers
loss of function
lung
Lung disease
Macrophage
mononuclear cell
mononuclear cells
Mortality
neutrophil
normalized
Pathogenesis
pathology
Patient
Peripheral blood
potential therapeutic target
Prevalence
pro-inflammatory
profile
proinflammatory
promoted
promoter
Protein
S100A9
SARS-CoV-2
SARS-CoV-2-infected cell
secretion
severity of COVID-19
Signaling
subject
targeting
Treatment
Viremia
virus production
virus replication
Virus replication.
with COVID-19
β-catenin
[DOI] 10.1128/jvi.00967-22 PMC 바로가기
[DOI] 10.1128/jvi.00967-22 PMC 바로가기